Hyperreflexia in Guillain-Barre syndrome: relation with acute motor axonal neuropathy and anti-GM1 antibody

Hyperreflexia in Guillain-Barré syndrome: relation with acute motor axonal neuropathy and anti-GM1 antibody.

OBJECTIVES To investigate the incidence of hyperreflexia in patients with Guillain-Barré syndrome (GBS), and its relation with electrodiagnosis of acute motor axonal neuropathy (AMAN), antiganglioside GM1 antibody, and Campylobacter jejuni infection. It was reported that patients with AMAN in northern China often had hyperreflexia in the recovery phase. METHODS In 54 consecutive Japanese pati...

Guillain–Barre syndrome with hyperreflexia: A variant

Guillain-Barre syndrome (GBS) is a common cause of acute peripheral neuropathy and is characterized by hyporeflexia or areflexia. Hyperreflexia has been rarely reported with acute motor axonal neuropathy. A 10-year-old boy presented with asymmetrical weakness of upper and lower limbs and change of voice. Weakness progressed in the hospital with involvement of multiple cranial nerves, preserved ...

Hyperreflexia in acute motor axonal neuropathy

Acute motor axonal neuropathy (AMAN) is a type of GBS accompanied by peripheral axonal involvement where acute motor paralysis, loss of reflex or hyporeflexia, insignificant sensory loss, and albumino-cytologic disproportion in cerebrospinal f luid are seen with no demyelinating findings in electromyography (EMG).[1] Commonly known as acute inflammatory demyelinating polyneuropathy (AIDP), GBS’...

Detection of Anti-Ganglioside Antibodies in Guillain-Barre Syndrome

Acute Motor Conduction Block Neuropathy: Another Distinct Variant of Guillain-Barre Syndrome.

We describe a patient who developed progressive weakness in all limbs without sensory symptoms 4 weeks after upper respiratory system infection. Electrophysiological findings suggested a new variant of Guillain-Barré syndrome named "acute motor conduction block neuropathy". Electrophysiological studies were performed at admission, 12th and 28th weeks. At the 28th week, the clinical examination ...